As the surface characteristics influence directly the biocompatibility of the stents, a smooth surface obtained after electrochemical polishing can minimize thrombosis effectively and also has a potential beneficial effect on neointimal hyperplasia .
What does NIH stand for?
NIH stands for Neointimal Hyperplasia
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See other definitions of NIH
We have 74 other meanings of NIH in our Acronym Attic
- New International Greek Testament Commentary
- North Island Grid Upgrade (New Zealand)
- Nederlandse Indoor Golf Vereniging (Dutch: Dutch Indoor Golf Association; Netherlands)
- Now I've Got You
- Now I've Got You Son of A Bitch
- Now I've Got You You Son of A Bitch
- Nationaal Instituut voor Gezondheidsbevordering en Ziektepreventie (Dutch: National Institute for Health Promotion and Disease Prevention)
- National Institute of Hydrology (India)
- National Institutes of Health (US)
- Natural Infant Hygiene
- News in Health (National Institutes of Health)
- Not Invented Here
- National Institutes of Health Recombinant DNA Advisory Committee
- National Institutes of Health Collaborative Clinical Trials in Transplantation
- National Institutes of Health Chronic Prostatitis Symptom Index
- National Institutes of Health Rapid Access to Interventional Development
- National Institutes of Health Autism Coordinating Committee
- National Institutes of Health/National Cancer Institute
- Nickel Hydrogen
- National Inline Hockey Association (Canada)
Samples in periodicals archive:
It recently has been investigated as a treatment for PCI patients because of indications that it can reduce neointimal hyperplasia and smooth muscle-cell proliferation following stent placement, Dr.
Sustained expression of MCP-1 may be more important than VCAM-1 in the process of neointimal hyperplasia featured by predominant SMC in the CAL mouse model since MCP-1 may both trigger the initial influx of leukocytes into the vessel wall and help to perpetuate the development of the intimal lesion by stimulating the proliferation and migration of SMC in intima.
E2F is a transcription factor family that is responsible for the activation of genes involved in the growth and proliferation of the smooth muscle cells that can lead to neointimal hyperplasia, plaque build-up, venous graft failure or restenosis.