DIM stimulated ERbeta but not ERalpha activation of an estrogen response element upstream of the luciferase reporter gene.
What does ERE stand for?
ERE stands for Estrogen Response Element
This definition appears very frequently and is found in the following Acronym Finder categories:
- Science, medicine, engineering, etc.
See other definitions of ERE
We have 93 other meanings of ERE in our Acronym Attic
- Energize Regional Economies
- Energy Resource Efficiency
- Energy Resources Engineering (various schools)
- Energy, Resources and the Environment (European Geosciences Union)
- English Rock Ensemble (band)
- Environmental Remediation Equipment
- Environmental Requirements Engineer
- Environmental Research and Education
- Environmental Research Event
- Espace Rural et Environnement (French: Environment and Rural Areas)
- Etheric Rain Engineering
- Evenly Rotating Economy (model)
- Extended Regular Expression
- Extensional Rheology Experiment
- Extra Regimental Employment
- Association of European Research Establishments in Aeronautics
- Edmund Rice Education Australia (Richmond, Victoria, Australia)
- Établissement Régional d'Enseignement Adapté (French: Adapted Regional Education Establishment)
- Edmonton Real Estate Board (Canada)
Samples in periodicals archive:
Ishikawa/ER[alpha] stable cells were used to determine changes in estrogen response element (ERE)-mediated target gene expression or rapid action-mediated effects.
At least 14 of the 208 genes have known estrogen response elements, areas that indicate that they are directly regulated by estrogen through its receptor.
The estrogen response element (ERE)--thymidine kinase-luciferase reporter gene (ERE-tk-Luc) consists of a consensus ERE with a minimal thymidine kinase promoter driving firefly luciferase activity.
The mRNA of the progesterone receptor in Ishikawa cells is expressed in higher amounts in the presence of compounds that bind to the estrogen receptor and activate the estrogen response element found upstream from the DNA encoding the PR.
2] or estrogenic mimics bind to ERs, receptor dimerization and recruitment of transcriptional comodulators are initiated, and the hormone-receptor complex binds to the estrogen response element (ERE) and subsequently regulates transcription in an ordered and cyclic manner (Metivier et al.
Signal transduction through the ER requires a translocation of the hormone/ receptor complex to the nucleus, where it can bind to estrogen response elements that bestow estrogen sensitivity on target genes.