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A two-dimensional transthoracic echocardiographic and Doppler study demonstrated a cavernous right atrium, dilatation of the right ventricle with impaired systolic function, flattening of the ventricular septum in systole and diastole suggesting right ventricular pressure and volume overload, severe tricuspid regurgitation, mild to moderate pulmonic regurgitation, a dilated inferior vena cava without inspiratory collapse indicating severe right atrial hypertension, a pulmonary arterial pressure judged by Doppler to be >75/30 mm Hg, Doppler evidence of an elevated right ventricular end-diastolic pressure, and normal left-sided valves and chambers with a left-ventricular ejection fraction of >55%.
It has been reported that alterations in left ventricular end-diastolic pressure affect the velocity and the time of mitral late diastolic flow (33,34).
The low ventricular compliance elevates left ventricular end-diastolic pressures, promoting increased pulmonary vascular pressures and pulmonary edema.
Left ventricular end-diastolic pressure was 20 mmHg and peak- to-peak systolic aortic-left ventricular gradient was found to be 15 mmHg.
Highly elevated right ventricular end-diastolic pressure due to decreased right ventricular compliance by severe infarction resulted in shortening of ICT and the pseudonormalized right ventricular MPI.
Left ventriculogram showed an ejection fraction of 80%, midventricular systolic obliteration, elevated left ventricular end-diastolic pressure, and an akinetic apical chamber.