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METHODS: Experimental colitis was induced with 1 mL trinitrobenzene sulfonic acid (TNBS) in 40% ethanol by intracolonic administration with 8-cm-long cannula under ether anesthesia to rats in colitis group and colitis + black cumin oil group.
The aims of this study were to investigate, if the inflammation markers formed in trinitrobenzene sulphonic acid (TNBS)-induced colitis affect the gastric tissue and to determine the possible protective effects of orally administered NS seed oil on the gastric tissue.
RDEA119 was shown to reduce damage to colonic tissue in two different mouse models of colitis, murine trinitrobenzene sulfonic acid (TNBS) colitis model and murine dextran sulfate sodium (DSS) colitis model.
To test whether the relative amounts of essential fatty acids in the diet affects colitis-related damage, Natalia Nieto and her colleagues at the University of Granada in Spain induced ulcerative colitis in a group of rats by administering a substance known as trinitrobenzene sulfonic acid through a tube into the animals' colons.
The target compounds are trinitrotoluene (TNT), trinitrobenzene (TNB), (N)-trinitro-1, 35% triazacyclohexane (RDX), and other nitrobodies.
The study evaluated the efficacy of VB-201 in three mouse models of IBD: trinitrobenzene sulfonic acid (TNBS)-induced colitis, T cell-induced colitis and dextran sulfate sodium (DSS)-induced colitis.
Galanin in the trinitrobenzene sulfonic acid rat model of experimental colitis.
The study investigated the efficacy of NU206 in reducing the severity of colitis induced by dextran sulfate sodium (DSS), trinitrobenzene sulfonic acid (TNBS), and by non-steroidal anti-inflammatory drug (NSAID) treatment in interleukin (IL)-10-deficient mice.