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Topics covered include clinical features of Cockayne and related syndromes, the molecular basis and molecular diagnosis of CS transcription-coupled repair, CS Group B protein and chromatin structure, cell signaling, cell cycle defects and apoptosis in CS, and overlap with xeroderma pigmentosum and other progeroid syndromes.
Other pathways include the nucleotide-excision repair, mismatch repair, double-stranded break repair, and transcription-coupled repair pathways.
Recent studies have also demonstrated that the transcription-coupled repair mechanism can contribute to the repair of 8-OH-Gua in eukaryotes in vitro and in vivo (21, 22).