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Samples in periodicals archive:
Angiotensin II-induced cardiomyocyte hypertrophy and cardiac fibrosis in stroke-prone spontaneously hypertensive rats.
Brief treatment with the ACE inhibitor delapril or the angiotensin II receptor antagonist candesartan significantly lowered blood pressure and suppressed development of proteinuria and nephrosclerosis in stroke-prone spontaneously hypertensive rats, compared with those treated with placebo or the vasodilator hydralazine (J.
Key words: Choto-san, stroke-prone spontaneously hypertensive rat, endothelium-dependent relaxation, [NO.