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Pulmonary sequestration was first described by Rektorzik in 1861, as a nonfunctioning mass of lung tissue which lacks normal communication with the tracheobronchial tree and with an anomalous systemic arterial supply.
Approximately 30% of patients with pericardial agenesis have additional anomalies such as atrial septal defect, bicuspid aortic valve, patent ductus arteriosus, tetralogy of Fallot, pulmonary sequestration, bronchogenic cyst and congenital diaphragmatic hernia (2, 3).
This factor is known to play a significant role in the frequent misdiagnoses of pulmonary hamartoma (18) and in rare cases of pulmonary sequestration (15); in both lesions, the culprit is the abundant benign cohesive epithelium often misinterpreted as well-differentiated adenocarcinoma.
Abstract: Pulmonary sequestration is a rare congenital or acquired pulmonary anomaly.
Long-term cardiovascular consequences of undiagnosed intralobar pulmonary sequestration.
This results in the pulmonary sequestration of PMNs.