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Samples in periodicals archive:
QT Prolongation and Proarrhythmia: SOTYLIZE can cause serious ventricular arrhythmias, primarily Torsade de Pointes (TdP) type ventricular tachycardia, a polymorphic ventricular tachycardia associated with QT interval prolongation.
Mapping and ablation of polymorphic ventricular tachycardia after myocardial infarction.
Quite simply, we demonstrate that estrogen promotes major cardiac events - such as polymorphic ventricular tachycardia (pVT) and SCD - while progesterone prevents them when LQT2 is a factor.
Mutations in the human RYR2 gene have been associated with two inherited cardiac diseases: arrhythmogenic right ventricular cardiomyopathy type 2 (ARVD2; OMIM 600996) and stress-induced polymorphic ventricular tachycardia [ventricular tachycardia, stress-induced, polymorphic (VT-SIP); OMIM 604772] (5-10).
Cardiac channelopathies are rare, inherited heart conditions including Long QT Syndrome (LQTS), LQTS large Deletion Duplication, Short QT Syndrome (SQTS), Brugada Syndrome (BrS) and Catecholaminergic Polymorphic Ventricular Tachycardia (CPVT).
Importantly, no episodes of monomorphic or polymorphic ventricular tachycardia were seen.
Commonly prescribed drugs, such as antibiotics, psychotropic agents and histamine HI-receptor antagonists, can prolong cardiac repolarization and might trigger a polymorphic ventricular tachycardia called torsades de pointes (1).
The inherited cardiac channelopathies, including Long QT Syndrome (LQTS), Brugada Syndrome (BrS) and Catecholaminergic Polymorphic Ventricular Tachycardia (CPVT), are the cause of death for over 30% of Sudden Unexplained Deaths (SUDS) and 10-15% of autopsy-negative Sudden Infant Deaths (SIDS).