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The probable molecular mechanism hypothesized was that by mutating desmosomal protein impairs desmosomal assembly releasing free plakoglobin from the desmosomes, which could translocate into the nucleus and through competition with [beta]-catenin suppress signaling through the canonical Wnt/ [beta]-catenin-Tcf/Lef pathway.
Defining E-cadherin-associated protein complexes in epithelial cells: plakoglobin, beta- and gamma-catenin are distinct components.
 Pemphigus vulgaris is an autoimmune skin disease characterized by acantholytic skin lesions with autoantibodies directed against desmoglen, plakoglobins, and cadherin polypeptides of desmosomes and other cell-cell adhesion molecules.