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Sleep is supported by natural cycles of activity in the brain and consists of two basic states: rapid eye movement (REM) sleep and non-rapid eye movement (NREM) sleep.
Checks on brain electrical activity suggested that this process might be happening in a sleep phase between deep sleep, and dreaming sleep, called stage 2 non-rapid eye movement sleep, when fact-based memories are moved from "temporary storage" in the brain's hippocampus to another area called the pre-frontal cortex.
Those dreams could be from REM or from non-Rapid Eye Movement Sleep [NREM].
A 90-minute snooze mostly involves non-rapid eye movement, or NREM, sleep.
This cycle is active during sleep, too, creating the cycles of rapid eye movement (REM, dreaming) and non-rapid eye movement (NREM, nondreaming).
The disorder most often occurs at the point where the sleeper's brainwaves become larger and he or she moves into deep, non-rapid eye movement sleep.
The first randomized study of tiagabine (Gabitril) in patients with primary insomnia found tiagabine 8 mg and 12 mg doses significantly maintained and enhanced "deeper" non-rapid eye movement stages of sleep, and did not cause next-day residual sedation at 4 mg and 8 mg.
 Role of the ventilatory control system There are state-related differences in respiratory control between the sleep and wake states and between the five stages of sleep, most predominantly between stages 1 through 4 non-rapid eye movement (NREM) and rapid eye movement (REM) sleep.