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Samples in periodicals archive:
Genetic disorders, including familial combined hyperlipidemia and familial hypertriglyceridemia, also cause hypertriglyceridemia.
Their topics include how much evidence is enough, epidemiological aspects of lipid and lipoprotein levels in relation to cardiovascular diseases, whether raising HDL protects against atherosclerosis, the optimal treatment of patients with familial combined hyperlipidemia, managing lipid and lipoprotein disorders in children, the link between chronic inflammatory diseases and cardiovascular risk, dealing with inborn errors in cholesterol and bile acid biosynthesis, and when to fear statin interactions.
Newly identified apolipoprotein AV gene predisposes to high plasma triglycerides in familial combined hyperlipidemia.
This is particularly significant for patients with diabetes-associated hyperlipidemia and those with familial combined hyperlipidemia -- the most frequent genetic disorder of blood lipoprotein metabolism.
For example, many patients with familial combined hyperlipidemia (FCH) would meet the diagnostic criterion of LDL-C [greater than or equal to] 5 mmol/L (Table 1) and could confound results if not excluded.
By contrast, minor defects are almost invariably present when hypertriglyceridemia is evident, for example, in the insulin resistance syndrome or familial combined hyperlipidemia.
Familial combined hyperlipidemia (FCHL) is the commonest form of hereditary hyperlipidemia (1,2).
Increased IDL is also found in diseases associated with accelerated atherosclerosis, such as type III dyslipidemia, type 2 diabetes mellitus, end stage renal disease (ESRD), and familial combined hyperlipidemia.