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The aim of this study was to test the effect of treatment with AGE on brachial artery flow mediated endothelium-dependent dilation (FMD) and circulating markers of oxidative stress and systemic inflammation.
Endothelium-dependent dilation in the systemic arteries of asymptomatic subjects relates to coronary risk factors and their interaction.
Flow-mediated endothelium-dependent dilation (FMD) was calculated as the percent change in diameter 1 min after cuff release relative to the baseline diameter before cuff release.
Studies should develop and include new and improved objective measures that link the built environment to biology wherever appropriate, such as serum glucose, endothelium-dependent dilation, cholesterol, insulin, energy balance, oxidative stress, body composition, and lipid profile.
Loss of flow-mediated endothelium-dependent dilation occurs early in the development of atherosclerosis.
In the first series of experiments, arteriolar endothelium-dependent dilation was evaluated in the four ROFA groups and the saline and Ti[O.
Impairment of endothelium-dependent dilation in rabbit renal arteries by oxidized lipoprotein(a).