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Indirect Immunofluorescence Assay Antibodies against ABV were detected by use of an IIFA on persistently Borna disease virus (BDV)-infected Madin-Darby canine kidney cells.
In The Neurobiology of Autism (Bauman and Kemper 2005), the authors draw parallels in neurobiology and behavior with autism and another type of infection, neonatal Borna disease virus.
Among the chapter topics are psychosocial and environmental formulations, monoaminergic-based pharmacotherapy, pyschobiology of electroshock, depression and suicidality in children and adolescents, the biology and genetics of suicidality, depression in the context of cancer, stress system dysregulation, neuroimmune mediators, Borna disease virus, animal models, and depression and heart disease.
Borna disease virus was named after the town of Borna in Saxony, southeastern Germany, where in 1885 many horses in a German cavalry regiment died of a fatal neurologic disease.
In Borna disease virus infected rabbit neurons 100 nm particle structures accumulate at areas of Joest-Degen inclusion bodies.
Borna disease virus (BDV) is the causative agent of Borna disease, a meningoencephalitis of horses and sheep in central Europe (1).
Future Research Directions and Perspective Although the detection of viruses in insectivores has been largely incidental or accidental, demonstration of Borna disease virus in brain tissues of the bicolored white-toothed shrew (Crocidura leucodon) (34) suggests that insectivores may play a greater role in the ecology of zoonotic diseases than previously appreciated.
Borna disease virus (BDV) is the causative agent of severe T-cell-mediated meningoencephalitis in horses, sheep, and other animal species in central Europe.