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The study by researchers in Australia also reveals the role of beta-amyloid precursor protein (APP), which forms plaques in affected brains.
Beta-Secretase (BACE-1) has been identified as a key enzyme that mediates a critical step in the formation of Beta-amyloid (ABeta40/42) by acting upon the Beta-amyloid precursor protein (Beta-APP).
While the mice given the beta-amyloid injections suffered no obvious ill effects, scientists caution that the shots could trigger an autoimmune response to the beta-amyloid precursor protein, which is present throughout the body.
Posiphen appears to modify the metabolism of beta-amyloid precursor protein (beta-APP).
Posiphen has been shown to lower beta-amyloid precursor protein (beta -APP) levels in pre-clinical studies.
The Phase IIB trial seeks to evaluate the potential ability of Phenserine to lower the levels of beta-amyloid precursor protein ((beta)-APP) and beta-amyloid (A(beta)) levels in the plasma and cerebrospinal fluid (CSF) in AD patients following six (6) months of treatment.
Lymphocytes of AD patients are impaired in their proliferative response to metabolic products of Beta-amyloid Precursor protein (APP).
Phenserine is currently undergoing testing in a Phase IIB trial to evaluate its anticipated ability to lower levels of beta-amyloid precursor protein ((beta)-APP) and beta-amyloid (A(beta)) in the plasma and cerebrospinal fluid (CSF), which may lead to a slowing of disease progression.