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Samples in periodicals archive:
lucidum spore protected against Cd(II)-induced liver injury in a dose-dependent manner, as evidenced by serum alanine aminotransferase, aspartate aminotransferase and histopathology.
On day 16 after the patient was admitted to the hospital, results of laboratory testing showed improved values for creatine phosphokinase (1,855 ng/mL), aspartate aminotransferase (101 IU/L), alanine aminotransferase (162 IU/L), lactate dehydrogenase (294 IU/L), and urine myoglobulin (10 [micro]g/L).
Serum iron, total iron binding capacity (TIBC), percentage saturation, body weight, albumin, aspartate aminotransferase, and hematocrit were monitored at regular intervals for 2.
Alanine aminotransferase (ALT) and aspartate aminotransferase (AST) levels were established using routine clinical chemistry testing.
The test, which is available from YorkTest Laboratories, can test for levels of the two enzymes, alanine aminotransferase (ALT) and aspartate aminotransferase (AST), which if found in high levels in the blood stream have been shown to increase higher risk of mortality.
Plasma alanine aminotransferase and aspartate aminotransferase activities were increased after TRI exposure in all mouse lines.
No associations were seen between microvesicular steatosis and lobular inflammation or levels of aspartate aminotransferase (AST) or alanine aminotransferase (ALT).
Urinalysis results, complete blood count, and potassium level were normal; complete metabolic panel results were unremarkable except for an elevated alanine aminotransferase level of 99 U/L (normal range, 21-72 U/L), aspartate aminotransferase level of 313 U/L (normal range, 17-59 U/L), chloride level of 108 mmol/L (normal range, 98-107 mmol/L), and total bilirubin level of 1.